Home / Longevity & Metabolic / 5-Amino-1MQ
PreclinicalLongevity & MetabolicNot a peptide

5-Amino-1MQ

A small-molecule NNMT inhibitor (not a peptide) · marketed online as a "fat loss without exercise" compound

Overview

5-Amino-1MQ is not a peptide, it is a small-molecule quinolinium compound that inhibits NNMT (nicotinamide N-methyltransferase), an enzyme upregulated in obese adipose tissue. In high-fat-diet mice it reduced fat mass and improved metabolic markers. There are no human trials. It is not approved as a medicine anywhere. We profile it because it is heavily marketed alongside peptides.

01 What is 5-Amino-1MQ?

In plain English.

5-Amino-1MQ is a small organic molecule, not a peptide. Its full chemical name is 5-amino-1-methylquinolinium, and it belongs to a chemical family called quinoliniums. It was designed by academic chemists at Indiana University as a selective inhibitor of an enzyme called NNMT (nicotinamide N-methyltransferase), which is unusually active in the fat tissue of obese mice and humans. The compound first appeared in the medicinal-chemistry literature in 2017–2018.

⏱ Half-life
Short (rat PK only)
☉ Route
Oral / IP (research)
⚖ Evidence
Preclinical (mouse)
📚 Studies
7 referenced

We include 5-Amino-1MQ in this peptide reference because it is sold and discussed in the same wellness, biohacker and "research-chemical" channels as peptides like MOTS-c, AOD-9604 and tesamorelin, and people regularly ask whether it is a peptide. It is not. It is a small molecule. The honest framing matters: peptide-adjacent marketing, peptide-style hype, but a different drug class with different risks.

02 How it works

The simple version, then the science.

NNMT's normal job is to mop up excess nicotinamide (a form of vitamin B3) by tagging it with a methyl group, which both removes it from the NAD+ salvage pathway and burns through SAM (the body's main methyl donor). In obesity, NNMT is dialled up in fat tissue, which the Kraus 2014 Nature paper argued helps drive a low-energy, fat-storing state. 5-Amino-1MQ blocks NNMT, the theory goes, so the cell stops wasting methyl groups and starts behaving more like lean tissue.

Go deeper · the proposed mechanism

Mechanistically, Neelakantan and colleagues at Indiana University (2017–2018) reported that 5-Amino-1MQ is a selective, cell- and membrane-permeable NNMT inhibitor with low-micromolar potency that increased intracellular NAD+ and SAM and reduced adipocyte size in 3T3-L1 cells and in diet-induced-obese mice. Kannt et al. (2018), working independently with a different small-molecule NNMT inhibitor, reproduced the broad picture: NNMT inhibition improved metabolic markers and reduced body weight gain in obese rodents. Pharmacokinetic work in rats (Patil et al. 2021) reported reasonable oral bioavailability and a short plasma half-life. None of this has been replicated in humans, and no human-subject data exists at the time of writing.

03 What it's used for

Each use graded by how strong the evidence actually is.

  • Preclinical
    Diet-induced obesity / adiposityIn Neelakantan et al. 2018 (Biochem Pharmacol), oral 5-Amino-1MQ for 11 days reduced body weight and fat mass in high-fat-diet mice without affecting food intake. This single mouse study is the cornerstone of essentially all consumer marketing.
  • Preclinical
    NAD+ / methyl-donor biologyInhibiting NNMT raises intracellular NAD+ and SAM in cell and rodent models, a mechanism plausibly relevant to metabolism and ageing biology, but only ever demonstrated in non-human systems.
  • Preclinical
    Broader metabolic markersA separate small-molecule NNMT inhibitor (Kannt 2018) reproduced weight and metabolic improvements in obese mice, lending some independent support to the target, but not to 5-Amino-1MQ specifically as a clinical drug.
  • Anecdotal
    "Fat loss without exercise" / wellness useSold and discussed in biohacker and peptide-clinic circles as a fat-loss compound. There are no controlled human trials in any indication. Every consumer claim extrapolates from rodent data.
Not a peptide. No human trials. Mouse data only. The "fat loss without exercise" framing comes from a single 11-day study in high-fat-diet mice. There is no published human-subject data on 5-Amino-1MQ, efficacy, dose, or safety.

04 What the evidence says

The evidence base for 5-Amino-1MQ is genuinely thin and entirely preclinical. The biology is interesting: NNMT was identified as an obesity-relevant enzyme by Kraus et al. in a 2014 Nature paper that knocked it down in fat tissue and protected mice from diet-induced obesity. That paper, not 5-Amino-1MQ, is the real foundation of the target. The Neelakantan group at Indiana University then developed quinolinium-based NNMT inhibitors, first reported in J Med Chem in 2017, then characterised in Biochem Pharmacol in 2018, where 5-Amino-1MQ reduced fat mass in high-fat-diet mice over an 11-day oral dosing period. An independent group at Sanofi/Roche (Kannt 2018, Sci Rep) reproduced metabolic improvements with a different small-molecule NNMT inhibitor, useful target validation, but not the same compound. A rat pharmacokinetics paper (Patil 2021) confirmed oral bioavailability. Honest read: a plausible, interesting target with a single short rodent study on this specific molecule, and a wellness market that has run far ahead of the data. We are still at "promising preclinical target", not "drug candidate that worked in people".

05 Dosing & administration

Reported in the literature, information not advice.

There is no human dose because there are no human trials. The mouse work used oral 5-Amino-1MQ at roughly 20 mg/kg/day for 11 days, which does not translate directly to a human dose. Online vendors and clinics quote daily oral capsule regimens in the tens-of-milligrams range, but these are not based on any controlled human pharmacokinetic or efficacy study. No safe or effective dose has been established in humans. A qualified clinician should be consulted before considering any unapproved compound.

06 Side effects & safety

Human safety data for 5-Amino-1MQ does not exist in any meaningful form, no published clinical trial, no pharmacovigilance system, no monitored population. That is the most important sentence on this page. Plausible mechanistic concerns include effects on NAD+ and methyl-donor (SAM) pools, which sit at the centre of metabolism and DNA methylation, a target where unknown effects on cancer, immune function and other tissues cannot be ruled out from rodent work alone. NNMT itself is expressed in many tissues beyond adipose, including liver and tumour cells, so chronic inhibition could plausibly affect those. Products sold as 5-Amino-1MQ are unregulated research chemicals with no requirement for identity, purity or dose accuracy. People who are pregnant, breastfeeding, immunocompromised, taking other medicines, or with any cancer history should be especially cautious.

Zero human safety data. Sold legally only as a research chemical, not for human consumption. The compound has never been through a published human trial of any size or duration.

07 Where to buy (research use only)

Vetted on quality and transparency, not an endorsement to use.

Helix Research Labs4.6
Research-use-only compounds with publicly available certificates of analysis. Listed for transparency, no purity standard addresses the underlying absence of human data on 5-Amino-1MQ.
HPLC & MS verifiedPublished COAsResearch use only
View ↗
Apex Compounds4.3
Competitive pricing across research chemicals. Listed for transparency, not an endorsement for human use.
Third-party testedResearch use only
View ↗
Vanta Bio4.5
Specialist supplier with independent lab testing. Product quality does not address the fact that no human has been monitored on this compound in a published study.
Independent lab testingResearch use only
View ↗
Disclosure: Pepwyse is not affiliated with these companies and does not earn any commission from these links; they are listed for reference only. These products are sold strictly for laboratory research use only and are not for human consumption.

09 Clinical studies & research

Primary sources. Read the science yourself.

Nicotinamide N-methyltransferase knockdown protects against diet-induced obesity
Nature · 2014 Animal (mouse) + cell
Kraus, Yang and colleagues showed that knocking down NNMT in white adipose tissue protected mice from high-fat-diet-induced obesity, raised NAD+ and SAM, and improved metabolic markers. The foundation paper underneath the entire NNMT-inhibition thesis, and the reason 5-Amino-1MQ exists. View on PubMed →
Selective and membrane-permeable small molecule inhibitors of nicotinamide N-methyltransferase reverse high fat diet-induced obesity in mice
Biochemical Pharmacology · 2018 Animal (mouse) + cell
Neelakantan et al. report that 5-Amino-1MQ (and a related quinolinium analogue) selectively inhibit NNMT, raise intracellular NAD+ and SAM, and, over 11 days of oral dosing, reduce fat mass and body weight in high-fat-diet mice without affecting food intake. This is the headline mouse study behind all 5-Amino-1MQ marketing. View on PubMed →
Structure-Activity Relationship for Small Molecule Inhibitors of Nicotinamide N-Methyltransferase
Journal of Medicinal Chemistry · 2017 Medicinal chemistry · in vitro
Neelakantan et al. describe the medicinal-chemistry programme that produced 5-amino-1-methylquinolinium-style NNMT inhibitors, the SAR work that identified the quinolinium scaffold and the 5-amino substitution as a tractable starting point. View on PubMed →
A small molecule inhibitor of Nicotinamide N-methyltransferase for the treatment of metabolic disorders
Scientific Reports · 2018 Animal (mouse) + cell
Kannt et al., independent of the Indiana group, report a separate small-molecule NNMT inhibitor that improved metabolic markers and reduced body-weight gain in obese mice. Useful target validation: it suggests the NNMT thesis is not specific to one chemical series. View on PubMed →
Nicotinamide N-Methyltransferase: More Than a Vitamin B3 Clearance Enzyme
Trends in Endocrinology and Metabolism · 2017 Review
Pissios review of NNMT biology, what the enzyme does, where it is expressed, why it is upregulated in obesity and cancer, and why it became a metabolic-drug target. Best single overview of why 5-Amino-1MQ exists. View on PubMed →
Nicotinamide N-methyltransferase: At the crossroads between cellular metabolism and epigenetic regulation
Molecular Metabolism · 2021 Review
Roberti, Ventura and Calza review NNMT across metabolism, ageing, cancer and epigenetics. Useful map of why inhibiting NNMT is plausibly interesting and why it is also not a one-tissue, one-effect target. View on PubMed →
Development & validation of LC-MS/MS assay for 5-amino-1-methyl quinolinium in rat plasma: Application to pharmacokinetic and oral bioavailability studies
Journal of Pharmaceutical and Biomedical Analysis · 2021 Animal (rat) · pharmacokinetics
Patil et al. characterise rat plasma pharmacokinetics and oral bioavailability of 5-Amino-1MQ. Useful technical work, but rat PK does not establish a safe or effective human dose. View on PubMed →

10 Frequently asked questions

Is 5-Amino-1MQ actually a peptide?
No. 5-Amino-1MQ is a small organic molecule, a quinolinium compound, not a peptide. We include it on this peptide reference because it is marketed and sold alongside peptides like MOTS-c and AOD-9604, and people often ask whether it is one. The distinction matters: peptides and small molecules have different absorption, distribution, regulatory pathways and risk profiles.
Does 5-Amino-1MQ really cause "fat loss without exercise"?
That claim comes from a single 11-day study in high-fat-diet mice (Neelakantan et al. 2018), in which oral 5-Amino-1MQ reduced fat mass without changing food intake. There are no controlled human trials showing the same effect in people. Treat the marketing slogan as a hypothesis from one mouse study, not a finding.
Have any humans taken 5-Amino-1MQ in a clinical trial?
No published human trial of 5-Amino-1MQ exists at the time of writing. The molecule has not been through a registered Phase 1, Phase 2 or Phase 3 study in any indication. Everything we know about its effects comes from cells, mice, and a rat pharmacokinetic study.
Is 5-Amino-1MQ approved by any regulator?
No. 5-Amino-1MQ is not approved as a medicine in the UK, US, EU, Australia or Canada. It is sold only as a research chemical, not for human consumption.
Is 5-Amino-1MQ banned in sport?
It is not named explicitly on the current WADA Prohibited List, but as an unapproved investigational substance with claimed performance/body-composition effects it can fall under S0 ("Non-Approved Substances"). Athletes in tested sport should treat it as prohibited.
Is 5-Amino-1MQ safe?
Unknown. There are no published human safety data. Because NNMT sits at the junction of NAD+, methyl-donor (SAM) pools and DNA methylation, and is expressed beyond fat in tissues including liver and tumour cells, the long-term consequences of chronic inhibition in humans cannot be predicted from short rodent studies. Products sold online are unregulated.
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